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ATCC human dlbcl cell line su dhl4

DGE inhibits cell proliferation and promotes DOX sensitivity in <t>DLBCL</t> cells. A : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of GM12878 cells; B : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of <t>SU-DHL4</t> cells; C : EdU assay to detect the effect of different concentrations of DGE on the proliferation rate of the cells; D : Colony formation assay to detect the effect of different concentrations of DGE on the ability of the cells to form clones; E : AnnexinV-PI double staining assay to detect the effect of different concentrations of DGE on the apoptosis rate of cells; F : CCK-8 assay to detect the effect of different concentrations of DGE on the sensitivity of SU-DHL4 cells to DOX. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Human Dlbcl Cell Line Su Dhl4, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 410 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human dlbcl cell line su dhl4 - by Bioz Stars, 2026-08

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1) Product Images from "Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis"

Article Title: Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis

Journal: Hereditas

doi: 10.1186/s41065-025-00531-3

DGE inhibits cell proliferation and promotes DOX sensitivity in DLBCL cells. A : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of GM12878 cells; B : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of SU-DHL4 cells; C : EdU assay to detect the effect of different concentrations of DGE on the proliferation rate of the cells; D : Colony formation assay to detect the effect of different concentrations of DGE on the ability of the cells to form clones; E : AnnexinV-PI double staining assay to detect the effect of different concentrations of DGE on the apoptosis rate of cells; F : CCK-8 assay to detect the effect of different concentrations of DGE on the sensitivity of SU-DHL4 cells to DOX. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Figure Legend Snippet: DGE inhibits cell proliferation and promotes DOX sensitivity in DLBCL cells. A : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of GM12878 cells; B : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of SU-DHL4 cells; C : EdU assay to detect the effect of different concentrations of DGE on the proliferation rate of the cells; D : Colony formation assay to detect the effect of different concentrations of DGE on the ability of the cells to form clones; E : AnnexinV-PI double staining assay to detect the effect of different concentrations of DGE on the apoptosis rate of cells; F : CCK-8 assay to detect the effect of different concentrations of DGE on the sensitivity of SU-DHL4 cells to DOX. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques Used: CCK-8 Assay, EdU Assay, Colony Assay, Clone Assay, Double Staining, Positive Control

DGE impairs metastasis and EMT in DLBCL cells. A - B : Transwell assay to detect the effect of different concentrations of DGE on cell migration and invasion ability; C : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular E-cadherin, N-cadherin and Vimentin. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Figure Legend Snippet: DGE impairs metastasis and EMT in DLBCL cells. A - B : Transwell assay to detect the effect of different concentrations of DGE on cell migration and invasion ability; C : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular E-cadherin, N-cadherin and Vimentin. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques Used: Transwell Assay, Migration, Western Blot, Expressing, Positive Control

DGE and DOX synergistically inhibit DLBCL cell growth in a mouse xenograft model. A : Schematic diagram of the in vivo experiment. Mice bearing tumors were treated with vehicle control, DGE (100 mg/kg/once per day, i.g.), DOX (1.5 mg/kg/once per week, i.p. ), or DGE + DOX for 21 days. n = 3 mice per group. B : Representative pictures of transplanted tumors in nude mice; C - D : The volume and weight of transplanted tumors in nude mice; E - F : Immunohistochemical assessment of the positive expression levels of Ki67 and N-cadherin proteins in the tissues of transplanted tumors in nude mice. * P < 0.05, ** P < 0.01, *** P < 0.001

Figure Legend Snippet: DGE and DOX synergistically inhibit DLBCL cell growth in a mouse xenograft model. A : Schematic diagram of the in vivo experiment. Mice bearing tumors were treated with vehicle control, DGE (100 mg/kg/once per day, i.g.), DOX (1.5 mg/kg/once per week, i.p. ), or DGE + DOX for 21 days. n = 3 mice per group. B : Representative pictures of transplanted tumors in nude mice; C - D : The volume and weight of transplanted tumors in nude mice; E - F : Immunohistochemical assessment of the positive expression levels of Ki67 and N-cadherin proteins in the tissues of transplanted tumors in nude mice. * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques Used: In Vivo, Control, Immunohistochemical staining, Expressing

DGE regulates mitochondrial function and glycolysis in DLBCL cells. A : MitoSOX Red staining to assess the effect of different concentrations of DGE on mitochondrial ROS; B : Flow cytometry analysis of the effect of different concentrations of DGE on MMP; C : Kits to detect the effects of different concentrations of DGE on cellular ATP production; D : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular PINK1 and PRKN; E - F : Kits to detect the effects of different concentrations of DGE on cellular glucose uptake and lactate production. Cells were treated with DGE for 24 h. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Figure Legend Snippet: DGE regulates mitochondrial function and glycolysis in DLBCL cells. A : MitoSOX Red staining to assess the effect of different concentrations of DGE on mitochondrial ROS; B : Flow cytometry analysis of the effect of different concentrations of DGE on MMP; C : Kits to detect the effects of different concentrations of DGE on cellular ATP production; D : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular PINK1 and PRKN; E - F : Kits to detect the effects of different concentrations of DGE on cellular glucose uptake and lactate production. Cells were treated with DGE for 24 h. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques Used: Staining, Flow Cytometry, Western Blot, Expressing

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Journal: Hereditas

Article Title: Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis

doi: 10.1186/s41065-025-00531-3

Figure Lengend Snippet: DGE inhibits cell proliferation and promotes DOX sensitivity in DLBCL cells. A : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of GM12878 cells; B : CCK-8 assay to detect the effect of different concentrations of DGE on the viability of SU-DHL4 cells; C : EdU assay to detect the effect of different concentrations of DGE on the proliferation rate of the cells; D : Colony formation assay to detect the effect of different concentrations of DGE on the ability of the cells to form clones; E : AnnexinV-PI double staining assay to detect the effect of different concentrations of DGE on the apoptosis rate of cells; F : CCK-8 assay to detect the effect of different concentrations of DGE on the sensitivity of SU-DHL4 cells to DOX. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Article Snippet: Normal human B lymphocytes (GM12878) with human DLBCL cell line SU-DHL4 (ATCC, VA, USA) were placed in Iscove’s modifed Dulbecco’s medium (Gibco, Grand Island, NY, USA) containing 10% fetal bovine serum (FBS, Gibco) and penicillin/streptomycin 100 U/mL and cultured at 37 °C, 5% CO 2 .

Techniques: CCK-8 Assay, EdU Assay, Colony Assay, Clone Assay, Double Staining, Positive Control

Journal: Hereditas

Article Title: Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis

doi: 10.1186/s41065-025-00531-3

Figure Lengend Snippet: DGE impairs metastasis and EMT in DLBCL cells. A - B : Transwell assay to detect the effect of different concentrations of DGE on cell migration and invasion ability; C : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular E-cadherin, N-cadherin and Vimentin. Cells were treated with DGE or DOX for 24 h, DOX (0.5 µM, 24 h) was used as a positive control. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques: Transwell Assay, Migration, Western Blot, Expressing, Positive Control

Journal: Hereditas

Article Title: Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis

doi: 10.1186/s41065-025-00531-3

Figure Lengend Snippet: DGE and DOX synergistically inhibit DLBCL cell growth in a mouse xenograft model. A : Schematic diagram of the in vivo experiment. Mice bearing tumors were treated with vehicle control, DGE (100 mg/kg/once per day, i.g.), DOX (1.5 mg/kg/once per week, i.p. ), or DGE + DOX for 21 days. n = 3 mice per group. B : Representative pictures of transplanted tumors in nude mice; C - D : The volume and weight of transplanted tumors in nude mice; E - F : Immunohistochemical assessment of the positive expression levels of Ki67 and N-cadherin proteins in the tissues of transplanted tumors in nude mice. * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques: In Vivo, Control, Immunohistochemical staining, Expressing

Journal: Hereditas

Article Title: Damnacanthus giganteus extract block diffuse large b-cell lymphoma proliferation and EMT by regulating mitochondrial dysfunction and glycolysis

doi: 10.1186/s41065-025-00531-3

Figure Lengend Snippet: DGE regulates mitochondrial function and glycolysis in DLBCL cells. A : MitoSOX Red staining to assess the effect of different concentrations of DGE on mitochondrial ROS; B : Flow cytometry analysis of the effect of different concentrations of DGE on MMP; C : Kits to detect the effects of different concentrations of DGE on cellular ATP production; D : Western blot to detect the effect of different concentrations of DGE on the expression levels of cellular PINK1 and PRKN; E - F : Kits to detect the effects of different concentrations of DGE on cellular glucose uptake and lactate production. Cells were treated with DGE for 24 h. Each group of experiments was repeated independently 3 times, * P < 0.05, ** P < 0.01, *** P < 0.001

Techniques: Staining, Flow Cytometry, Western Blot, Expressing

The bulk m6A RNA methylation and METTL3 expression are increased in DLBCL. (A) The bulk m6A RNA methylation in 18 DLBCL tissues and 18 inflammatory lymph glands. ∗ P < 0.05. (B) The bulk m6A RNA methylation in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (C) mRNA expression of METTL3 in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗∗ P < 0.01. (D) METTL3 expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (E) qRT-PCR was used to analyze mRNA expression of METTL3 in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (F) METTL3 expression in 4 DLBCL tissues and 4 inflammatory lymph glands was analyzed by western blotting. ∗∗ P < 0.01. (G) mRNA expression of METTL14 in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗∗ P < 0.01. (H) qRT-PCR was used to analyze mRNA expression of METTL14 in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (I) METTL14 expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (J) mRNA expression of WTAP in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗ P < 0.05. (K) qRT-PCR was used to analyze mRNA expression of WTAP in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗ P < 0.05. (L) WATP expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05.

Journal: Frontiers in Genetics

Article Title: The m6A Methyltransferase METTL3 Is Functionally Implicated in DLBCL Development by Regulating m6A Modification in PEDF

doi: 10.3389/fgene.2020.00955

Figure Lengend Snippet: The bulk m6A RNA methylation and METTL3 expression are increased in DLBCL. (A) The bulk m6A RNA methylation in 18 DLBCL tissues and 18 inflammatory lymph glands. ∗ P < 0.05. (B) The bulk m6A RNA methylation in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (C) mRNA expression of METTL3 in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗∗ P < 0.01. (D) METTL3 expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (E) qRT-PCR was used to analyze mRNA expression of METTL3 in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (F) METTL3 expression in 4 DLBCL tissues and 4 inflammatory lymph glands was analyzed by western blotting. ∗∗ P < 0.01. (G) mRNA expression of METTL14 in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗∗ P < 0.01. (H) qRT-PCR was used to analyze mRNA expression of METTL14 in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗∗ P < 0.01. (I) METTL14 expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (J) mRNA expression of WTAP in 18 DLBCL tissues and 18 inflammatory lymph glands was analyzed by qRT-PCR. ∗ P < 0.05. (K) qRT-PCR was used to analyze mRNA expression of WTAP in DLBCL cell lines (SU-DHL4, OCILy10, Farage, U2932, HBL1) and Human B lymphocyte (GM12878). ∗ P < 0.05. (L) WATP expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05.

Article Snippet: Human DLBCL cell lines SU-DHL4, OCILy10, Farage, U2932, and HBL1 as well as human B lymphocyte GM12878 cell line were obtained from the ATCC.

Techniques: Methylation, Expressing, Quantitative RT-PCR, Western Blot

METTL3 knockdown suppresses the proliferation of DLBCL cells. qRT-PCR (A) and western blotting (B) were used to assess the efficacy of METTL3 silencing in SU-DHL4 and HBL1 cells, respectively. ∗∗ P < 0.01. The viability of the DLBCL cells was determined using CCK8 (C) and MTT (D) assays. (E) The cell cycle distribution of the DLBCL cells was detected by flow cytometry. ∗ P < 0.05. (F) Apoptosis in SU-DHL4 and HBL1 cells was examined by using Annexin V assay. ∗ P < 0.05.

Journal: Frontiers in Genetics

Article Title: The m6A Methyltransferase METTL3 Is Functionally Implicated in DLBCL Development by Regulating m6A Modification in PEDF

doi: 10.3389/fgene.2020.00955

Figure Lengend Snippet: METTL3 knockdown suppresses the proliferation of DLBCL cells. qRT-PCR (A) and western blotting (B) were used to assess the efficacy of METTL3 silencing in SU-DHL4 and HBL1 cells, respectively. ∗∗ P < 0.01. The viability of the DLBCL cells was determined using CCK8 (C) and MTT (D) assays. (E) The cell cycle distribution of the DLBCL cells was detected by flow cytometry. ∗ P < 0.05. (F) Apoptosis in SU-DHL4 and HBL1 cells was examined by using Annexin V assay. ∗ P < 0.05.

Article Snippet: Human DLBCL cell lines SU-DHL4, OCILy10, Farage, U2932, and HBL1 as well as human B lymphocyte GM12878 cell line were obtained from the ATCC.

Techniques: Knockdown, Quantitative RT-PCR, Western Blot, Flow Cytometry, Annexin V Assay

$METTL3 knockdown down-regulates PEDF expression and m6A methylation in PEDF mRNAs, as well as Wnt signaling activities. (A) PEDF expression and correlation of the expression of METTL3 with PEDF expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (B) mRNA expression of PEDF in 18 DLBCL tissues and 18 inflammatory lymph gland specimens was determined using qRT-PCR. ∗∗ P < 0.01. A positive correlation between mRNA expression of METTL3 and PEDF was detected by linear regression analysis. qRT-PCR (C) and western blotting (D) were used to analyze mRNA and protein expression of PEDF in SU-DHL4 and HBL1 cells with silenced expression of METTL3, respectively. ∗∗ P < 0.01. TOP/FOP-Flash reporter (E) was employed to determine Wnt signaling activity in SU-DHL4 and HBL1 cells with silenced expression of METTL3. (F) Western blotting assay of total and nuclear β-catenin proteins in DLBCL cells with silenced expression of METTL3. GAPDH and Lamin B1 were used as internal control and endogenous control of cell nuclear fraction, respectively. (G) Accumulation of β-catenin in the nucleus of the DLBCL cells with silenced expression of METTL3 according to confocal microscope images. (H) Western blotting assay of total and phosphorylated LRP6 proteins in DLBCL cells with silenced expression of METTL3. GAPDH was used as internal control. Me-RIP (I) assay was conducted to determine m6A methylation in PEDF transcripts in SU-DHL4 and HBL1 cells with silenced expression of METTL3. ∗∗ P < 0.01. (J) The half-life (T1/2) of PEDF mRNAs in SU-DHL4 and HBL1 cells transfected with Lv-shMETTL3 or Lv-NC (the control lentivirus).$

Journal: Frontiers in Genetics

Article Title: The m6A Methyltransferase METTL3 Is Functionally Implicated in DLBCL Development by Regulating m6A Modification in PEDF

doi: 10.3389/fgene.2020.00955

Figure Lengend Snippet: METTL3 knockdown down-regulates PEDF expression and m6A methylation in PEDF mRNAs, as well as Wnt signaling activities. (A) PEDF expression and correlation of the expression of METTL3 with PEDF expression in TCGA database between DLBCL tissues and normal counterparts. ∗ P < 0.05. (B) mRNA expression of PEDF in 18 DLBCL tissues and 18 inflammatory lymph gland specimens was determined using qRT-PCR. ∗∗ P < 0.01. A positive correlation between mRNA expression of METTL3 and PEDF was detected by linear regression analysis. qRT-PCR (C) and western blotting (D) were used to analyze mRNA and protein expression of PEDF in SU-DHL4 and HBL1 cells with silenced expression of METTL3, respectively. ∗∗ P < 0.01. TOP/FOP-Flash reporter (E) was employed to determine Wnt signaling activity in SU-DHL4 and HBL1 cells with silenced expression of METTL3. (F) Western blotting assay of total and nuclear β-catenin proteins in DLBCL cells with silenced expression of METTL3. GAPDH and Lamin B1 were used as internal control and endogenous control of cell nuclear fraction, respectively. (G) Accumulation of β-catenin in the nucleus of the DLBCL cells with silenced expression of METTL3 according to confocal microscope images. (H) Western blotting assay of total and phosphorylated LRP6 proteins in DLBCL cells with silenced expression of METTL3. GAPDH was used as internal control. Me-RIP (I) assay was conducted to determine m6A methylation in PEDF transcripts in SU-DHL4 and HBL1 cells with silenced expression of METTL3. ∗∗ P < 0.01. (J) The half-life (T1/2) of PEDF mRNAs in SU-DHL4 and HBL1 cells transfected with Lv-shMETTL3 or Lv-NC (the control lentivirus).

Article Snippet: Human DLBCL cell lines SU-DHL4, OCILy10, Farage, U2932, and HBL1 as well as human B lymphocyte GM12878 cell line were obtained from the ATCC.

Techniques: Knockdown, Expressing, Methylation, Quantitative RT-PCR, Western Blot, Activity Assay, Control, Microscopy, Transfection

The overexpression of PEDF reverses the inhibitory effects of METTL3 knockdown on DLBCL cell proliferation. PEDF expression at mRNA (A) and protein levels (B) was determined using qRT-PCR and western blot analysis, respectively. CCK8 (C) and Annexin V assays (D) were employed to detect the viability and apoptotic rate of SU-DHL4 and HBL1 cells, respectively. (E) The proliferative activity of SU-DHL4 and HBL1 cells in the mouse model was assessed. Bioluminescence images of the mice bearing SU-DHL4-luc and HBL1-luc cells as well as relative luminescence intensities were illustrated. All experiments above were conducted after transfection with Lv-NC, Lv-shMETTL3, Lv-PEDF and Lv-shMETTL3 + Lv-PEDF. ∗ P < 0.05, ∗∗ P < 0.01 vs. the control groups.

Journal: Frontiers in Genetics

Article Title: The m6A Methyltransferase METTL3 Is Functionally Implicated in DLBCL Development by Regulating m6A Modification in PEDF

doi: 10.3389/fgene.2020.00955

Figure Lengend Snippet: The overexpression of PEDF reverses the inhibitory effects of METTL3 knockdown on DLBCL cell proliferation. PEDF expression at mRNA (A) and protein levels (B) was determined using qRT-PCR and western blot analysis, respectively. CCK8 (C) and Annexin V assays (D) were employed to detect the viability and apoptotic rate of SU-DHL4 and HBL1 cells, respectively. (E) The proliferative activity of SU-DHL4 and HBL1 cells in the mouse model was assessed. Bioluminescence images of the mice bearing SU-DHL4-luc and HBL1-luc cells as well as relative luminescence intensities were illustrated. All experiments above were conducted after transfection with Lv-NC, Lv-shMETTL3, Lv-PEDF and Lv-shMETTL3 + Lv-PEDF. ∗ P < 0.05, ∗∗ P < 0.01 vs. the control groups.

Article Snippet: Human DLBCL cell lines SU-DHL4, OCILy10, Farage, U2932, and HBL1 as well as human B lymphocyte GM12878 cell line were obtained from the ATCC.

Techniques: Over Expression, Knockdown, Expressing, Quantitative RT-PCR, Western Blot, Activity Assay, Transfection, Control

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